Friday, January 17, 2014

Taiwan Opening to Canadian beef conditional: Health Ministry BSE CJD UPDATE

Opening to Canadian beef conditional: Health Ministry 2014/01/17 17:30:08

 

Taipei, Jan. 17 (CNA) Taiwan's upcoming market-opening measure for Canadian beef will not cover cattle parts with a high risk of carrying mad cow disease, the Ministry of Health and Welfare said Friday.

 

These high-risk parts include internal organs, skulls, eyes, brains, spinal cords and ground meat, which will remain barred from entering Taiwan, said Chiang Yu-mei, deputy director-general of the Food and Drug Administration under the ministry.

 

The Ministry of Economic Affairs announced earlier in the day that the government is to allow imports of Canadian bone-in beef and other meat products from cattle under 30 months of age.

 

At present, only boneless beef from cattle under 30 months of age is allowed into the country.

 

Chiang noted that Canada, which last reported a bovine spongiform encephalopathy (BSE) outbreak three years ago, is currently listed as a country with "controlled BSE risk" by the World Organization for Animal Health.

 

The decision to open Taiwan's market to Canadian beef was made after a risk assessment, discussions and four inspection tours of the North American country's cattle industry, with a focus on feed and slaughterhouse sanitation, she said.

 

The conditions set by Taiwan for Canadian beef imports will be the same as those for beef from the United States, another BSE-affected country, she added.

 

Based on data from 2012, as much as 94 percent of beef demand in Taiwan is met by imports, with 46 percent from Australia, 24.76 percent from the United States, 23 percent from New Zealand and 0.3 percent from Canada. Other smaller suppliers include Panama, Nicaragua and Costa Rica.

 

Agriculture Minister Chen Bao-ji said the opening will have little impact on Taiwan's cattle industry, which has only a 6 percent market share.

 

Agreeing with Chen, Chang Chuan-chung, chairman of the Taiwan Beef Industry Progress Association, said domestically produced beef is well-received among local customers and enjoys a stable market share because Taiwan's cattle industry does not use leanness enhancing drugs in raising the animals.

 

Legislators, meanwhile, expressed their support for the plan.

 

Legislative Yuan Speaker Wang Jin-pyng said the conditional opening of Canadian beef imports takes into account the safety of consumers while demonstrating Taiwan's commitment to fair trade.

 

The move should be helpful to Taiwan's bid to join the Trans-Pacific Partnership, he said.

 

Hsu Chung-hsin of the opposition Taiwan Solidarity Union said the opening falls in line with the most-favored-nation principle of the World Trade Organization, given that Taiwan has already opened its market to U.S. bone-in beef.

 

(By Lung Jui-yun, Yang Shu-min, Chen Wei-ting and Y.F. Low) ENDITEM/J

 

 


 

 

> Hsu Chung-hsin of the opposition Taiwan Solidarity Union said the opening falls in line with the most-favored-nation principle of the World Trade Organization, given that Taiwan has already opened its market to U.S. bone-in beef.

 

 IN reality, this does fall in line with the WTO/OIE/USDA inc mad cow BSE trade policy. exactly what is does, it allows Transmissible Spongiform Encephalopathy TSE prion disease to be accepted as part of the commodity itself. yes, this allows the free trading of the Transmissible Spongiform Encephalopathy TSE prion mad cow type disease. what it also does now, it has made it acceptable for death to come from different commodities, if the _documented_ body bag count does not get to high

 

(key word here is documented, because if it all stays sporadic, problem solved $$$, remember, all iatrogenic CJD is, is sporadic CJD, until the route and source of the iatrogenic event that took places is proven, documented, and then put in the academic domain, which very rarely ever happens do to the long incubation, and then trace back there from),

 

and IF, the trace back to any friendly fire cannot be proven, then the BSE MMR is what it is, minimal risk, which means that some risk is acceptable. this now includes the iatrogenic (friendly fire, pass it forward, medical, surgical, dental, tissue, blood) spread of the TSE prion mad cow type disease around the globe, and you never have to leave home. just think about that for a while.

 

kind regards, terry

 

 

 

IN A NUT SHELL ;

 

(Adopted by the International Committee of the OIE on 23 May 2006)

 

11. Information published by the OIE is derived from appropriate declarations made by the official Veterinary Services of Member Countries.

 

*** The OIE is __not___ responsible for inaccurate publication of country disease status based on inaccurate information or changes in epidemiological status or other significant events that were not promptly reported to the Central Bureau,***

 


 

 

Sunday, December 15, 2013

 

*** FDA PART 589 -- SUBSTANCES PROHIBITED FROM USE IN ANIMAL FOOD OR FEED VIOLATIONS OFFICIAL ACTION INDICATED OAI UPDATE DECEMBER 2013 UPDATE

 


 

 

Saturday, December 21, 2013

 

**** Complementary studies detecting classical bovine spongiform encephalopathy infectivity in jejunum, ileum and ileocaecal junction in incubating cattle ****

 


 

 

Saturday, December 15, 2012

 

*** Bovine spongiform encephalopathy: the effect of oral exposure dose on attack rate and incubation period in cattle -- an update 5 December 2012

 


 

 

Monday, November 19, 2012

 

*** Prion in Saliva of Bovine Spongiform Encephalopathy–Infected Cattle

 




Subject: Infectivity in Skeletal Muscle of Cattle with Atypical Bovine Spongiform Encephalopathy

 

UPDATE NORTH AMERICA MAD COW DISEASE

 

Monday, March 19, 2012

 

Infectivity in Skeletal Muscle of Cattle with Atypical Bovine Spongiform Encephalopathy

 

PLoS One. 2012; 7(2): e31449.

 


 

 

USDA AUDIT ON CANADA'S MEAT INSPECTION DISTURBING (pot calling kettle black again)

 

EDMONTON - Some of former Alberta premier Ralph Klein's most colourful quotes — and the reactions they elicited:

 

SNIP...

 

"This all came about through the discovery of a single, isolated case of mad cow disease in one Alberta cow on May 20th. The farmer — I think he was a Louisiana fish farmer who knew nothing about cattle ranching. I guess any self-respecting rancher would have shot, shovelled and shut up, but he didn't do that." — Klein recalls how the mad cow crisis started and rancher Marwyn Peaster's role. The premier was speaking at the Western Governors Association meeting in Big Sky, Mont. September 2004.

 

"The premier meant that in an ironic or almost a sarcastic way." — Klein spokesman Gordon Turtle.

 

---

 

"You would have to eat 10 billion meals of brains, spinal cords, ganglia, eyeballs and tonsils." — Klein speaking in Montreal in January 2005 on the risk of humans contracting mad cow disease.

 

---

 

"I would offer $5 billion to have a Japanese person to come over here and eat nothing but Alberta beef for a year. And if he gets mad cow disease, I would be glad to give him $5 billion — make it $10 billion — Canadian." — Klein speaking after Japan closed its borders to Canadian beef.

 

---

 


 

 

Wednesday, December 22, 2010.

 

Manitoba veterinarian has been fined $10,000 for falsifying certification documents for U.S. bound cattle and what about mad cow disease?

 


 

 

CENSORSHIP IS A TERRIBLE THING $$$.

 

Canada has had a COVER-UP policy of mad cow disease since about the 17th case OR 18th case of mad cow disease. AFTER THAT, all FOIA request were ignored $$$.

 

THIS proves there is indeed an epidemic of mad cow disease in North America, and it has been covered up for years and years, if not for decades, and it’s getting worse $$$.

 

Thursday, February 10, 2011.

 

TRANSMISSIBLE SPONGIFORM ENCEPHALOPATHY REPORT UPDATE CANADA FEBRUARY 2011 and how to hide mad cow disease in Canada Current as of: 2011-01-31.

 


 

 

Thursday, January 17, 2013.

 

Canada, U.S. agree on animal-disease measures to protect trade, while reducing human and animal health protection.

 


 

 

Wednesday, August 11, 2010.

 

REPORT ON THE INVESTIGATION OF THE SIXTEENTH CASE OF BOVINE SPONGIFORM ENCEPHALOPATHY (BSE) IN CANADA.

 


 

 

Thursday, August 19, 2010.

 

REPORT ON THE INVESTIGATION OF THE SEVENTEENTH CASE OF BOVINE SPONGIFORM ENCEPHALOPATHY (BSE) IN CANADA.

 


 

 

Friday, March 4, 2011.

 

Alberta dairy cow found with mad cow disease.

 


 

 

Increased Atypical Scrapie Detections.

 

Press reports indicate that increased surveillance is catching what otherwise would have been unreported findings of atypical scrapie in sheep. In 2009, five new cases have been reported in Quebec, Ontario, Alberta, and Saskatchewan. With the exception of Quebec, all cases have been diagnosed as being the atypical form found in older animals. Canada encourages producers to join its voluntary surveillance program in order to gain scrapie-free status. The World Animal Health will not classify Canada as scrapie-free until no new cases are reported for seven years. The Canadian Sheep Federation is calling on the government to fund a wider surveillance program in order to establish the level of prevalence prior to setting an eradication date. Besides long-term testing, industry is calling for a compensation program for farmers who report unusual deaths in their flocks.

 


 

 

Tuesday, May 21, 2013

 

Canada, USA, Bad feed, mad cows: Why we know three BSE cases had a common origin and why the SSS policy is in full force $$$

 


 

 

Friday, January 10, 2014

 

USDA AUDIT ON CANADA'S MEAT INSPECTION DISTURBING (pot calling kettle black again)

 


 

 

Wednesday, December 4, 2013

 

*** Bovine Spongiform Encephalopathy; Importation of Bovines and Bovine Products; Final Rule Federal Register / Vol. 78 , No. 233 / Wednesday, December 4, 2013

 


 

 

Saturday, November 2, 2013

 

*** APHIS Finalizes Bovine Import Regulations in Line with International Animal Health Standards while enhancing the spread of BSE TSE prion mad cow type disease around the Globe

 


 

 

Wednesday, January 15, 2014

 

*** INFECTION PREVENTION AND CONTROL OF CJD, VCJD AND OTHER HUMAN PRION DISEASES IN HEALTHCARE AND COMMUNITY SETTINGS Variably Protease-Sensitive Prionopathy (VPSPr) January 15, 2014

 


 

 

Wednesday, December 11, 2013

 

*** Detection of Infectivity in Blood of Persons with Variant and Sporadic Creutzfeldt-Jakob Disease

 


 


 

 

Monday, January 13, 2014

 

*** Prions in Variably Protease-Sensitive Prionopathy: An Update Pathogens 2013 Pathogens 2013, 2, 457-471; doi:10.3390/pathogens2030457

 


 

 

Friday, January 10, 2014

 

*** vpspr, sgss, sffi, TSE, an iatrogenic by-product of gss, ffi, familial type prion disease, what it ???

 


 

 

*** PRICE OF CWD TSE PRION POKER GOES UP 2014 ***

 

Wednesday, January 01, 2014

 

Molecular Barriers to Zoonotic Transmission of Prions

 

*** chronic wasting disease, there was no absolute barrier to conversion of the human prion protein.

 

*** Furthermore, the form of human PrPres produced in this in vitro assay when seeded with CWD, resembles that found in the most common human prion disease, namely sCJD of the MM1 subtype.

 


 


 

 

Saturday, November 16, 2013

 

Management of neurosurgical instruments and patients exposed to creutzfeldt-jakob disease 2013 December

 

Infect Control Hosp Epidemiol.

 


 

 

Thursday, November 14, 2013

 

Prion diseases in humans: Oral and dental implications

 


 

 

Saturday, November 2, 2013

 

Recommendation of the Swiss Expert Committee for Biosafety on the classification of activities using prion genes and prion protein January 2013

 


 


 

 

Wednesday, December 11, 2013

 

*** Detection of Infectivity in Blood of Persons with Variant and Sporadic Creutzfeldt-Jakob Disease

 


 


 

 

Monday, January 13, 2014

 

*** Prions in Variably Protease-Sensitive Prionopathy: An Update Pathogens 2013 Pathogens 2013, 2, 457-471; doi:10.3390/pathogens2030457

 


 

 

Friday, January 10, 2014

 

*** vpspr, sgss, sffi, TSE, an iatrogenic by-product of gss, ffi, familial type prion disease, what it ???

 


 

 

*** PRICE OF CWD TSE PRION POKER GOES UP 2014 ***

 

 

Wednesday, January 01, 2014

 

Molecular Barriers to Zoonotic Transmission of Prions

 

*** chronic wasting disease, there was no absolute barrier to conversion of the human prion protein.

 

*** Furthermore, the form of human PrPres produced in this in vitro assay when seeded with CWD, resembles that found in the most common human prion disease, namely sCJD of the MM1 subtype.

 


 


 

 

Saturday, November 16, 2013

 

Management of neurosurgical instruments and patients exposed to creutzfeldt-jakob disease 2013 December

 

Infect Control Hosp Epidemiol.

 


 

 

Thursday, November 14, 2013

 

Prion diseases in humans: Oral and dental implications

 


 

 

Saturday, November 2, 2013

 

Recommendation of the Swiss Expert Committee for Biosafety on the classification of activities using prion genes and prion protein January 2013

 


 

 

Sunday, August 11, 2013

 

Creutzfeldt-Jakob Disease CJD cases rising North America updated report August 2013

 

*** Creutzfeldt-Jakob Disease CJD cases rising North America with Canada seeing an extreme increase of 48% between 2008 and 2010

 


 

 

Sunday, October 13, 2013

 

*** CJD TSE Prion Disease Cases in Texas by Year, 2003-2012

 


 

 

Terry S. Singeltary Sr. on the Creutzfeldt-Jakob Disease Public Health Crisis

 

 

TAIWAN SupremeMASTER

 

 


 

 


 

 


 

 

full text with source references ;

 

 


 

 

 

 

News

 

Taiwan

 

CDC

 

updates

 

Creutzfeldt-Jakob disease (CJD) incidence in Taiwan and announces two new highly probable cases of sporadic Creutzfeldt-Jakob disease (sCJD)

 

According to the result of the third case investigation in 2011 conducted by the Creutzfeldt-Jakob disease (CJD) working group of the Taiwan Neurological Society, the Taiwan Centers for Disease Control (Taiwan CDC) announces the latest updates on the Creutzfeldt-Jakob disease (CJD) surveillance data and reports two highly probable cases of sporadic Creutzfeldt-Jakob disease (sCJD). Between 1997 and March of 2011, a total of 439 suspected CJD cases were reported and investigated in Taiwan, including 259 probable, highly probable and confirmed CJD cases. Of the 259 cases, 255 cases are sCJD cases, including 27 probable, 226 highly probable and 2 confirmed sCJD cases, 3 cases are confirmed genetic CJD (gCJD) cases, and one case is a highly probable variant CJD (vCJD) case imported from the UK. Over the past decade, the annual CJD incidence rate in Taiwan is approximately 0.5~1 case per 1,000,000 persons, which is similar to that in other countries around the world.

 

There are four types of CJD: sporadic Creutzfeldt-Jakob disease (sCJD), acquired CJD, genetic CJD and bovine spongiform encephalopathy (BSE)-related variant CJD (vCJD). sCJD accounts for about 80~90% of all CJD cases. sCJD is believed to result from the build-up of abnormal prion proteins. Acquired CJD is usually transmitted through certain medical procedures such as injections of contaminated pituitary growth hormone and grafts of contaminated dura mater.

 

Taiwan CDC states that CJD is listed as one of the Category IV Notifiable Infectious Diseases in Taiwan and Taiwan has a comprehensive approach to managing CJD, including a case reporting system, a disease surveillance system, a case investigation mechanism, nosocomial infection control guidelines, and case reporting guidelines. When a probable, highly probable or confirmed case is reported, Taiwan CDC will request the Taiwan Blood Services Foundation, the Bureau of Medical Affairs and the diagnosing hospital in writing to tighten their implementation of blood safety strategies, graft control measures and nosocomial transmission and infection control measures to reduce the risk of transmission. For more information on CJD, please visit the Taiwan CDC’s website: http://www.cdc.gov.tw/

 

 

Hit: 2125 Updated: 2012-4-10 11:20 Reviewed: 2013-11-19 11:34 Source: Taipei City Hospital

 

 


 

 

 

Sunday, May 27, 2012

 

Taiwan Group urges halt of U.S. beef imports due to 'L-type' mad cow disease

 


 

 

 

 

Tuesday, February 15, 2011

 

TAIWAN DOH censured for downplaying probable human mad-cow disease death 2011/02/15 18:32:27

 


 

 

 

 

 

TSS

 

 

with kindest regards, terry

 

Terry S. Singeltary Sr.

 

flounder9@verizon.net

 

 

Sunday, May 27, 2012

Taiwan Group urges halt of U.S. beef imports due to 'L-type' mad cow disease

Taiwan Group urges halt of U.S. beef imports due to 'L-type' mad cow disease


2012/05/27 18:11:07 Taipei, May 27


(CNA) The Consumers' Foundation called Sunday for a halt to imports of U.S. beef following a report by an American consumer group that a recent U.S. mad cow disease case was an "L-type" atypical strain, which it said can be transmitted to humans.










What irks many scientists is the USDA’s April 25 statement that the rare disease is “not generally associated with an animal consuming infected feed.”



The USDA’s conclusion is a “gross oversimplification,” said Dr. Paul Brown, one of the world’s experts on this type of disease who retired recently from the National Institutes of Health.

"(The agency) has no foundation on which to base that statement.”



“We can’t say it’s not feed related,” agreed Dr. Linda Detwiler, an official with the USDA during the Clinton Administration now at Mississippi State. In the May 1 email to me, USDA’s Cole backed off a bit. “No one knows the origins of atypical cases of BSE,” she said


Few scientists would argue that the one California cow which never was headed to the U.S. food supply represents a health hazard.



But many maintain that the current surveillance is insufficient.



Dr. Kurt Giles, an expert in neurogenerative diseases now at the University of California, San Francisco, was at Oxford during the British outbreak.

He told me USDA’s assurances about safety today remind him of British statements during the 1980s.



“It is so reminiscent of that absolute certainty,” he said.



Robert Bazell is NBC's chief science and medical correspondent. Follow him on Facebook and on Twitter @RobertBazellNBC








Saturday, May 26, 2012



Are USDA assurances on mad cow case 'gross oversimplification'?







Friday, May 25, 2012


R-CALF USDA’s New BSE Rule Eliminates Important Protections Needed to Prevent BSE Spread







in the url that follows, I have posted SRM breaches first, as late as 2011.



NEXT



MAD COW FEED BAN BREACHES AND TONNAGES OF MAD COW FEED IN COMMERCE up until 2007, when they ceased posting them.



NEXT



MAD COW SURVEILLANCE BREACHES.



Friday, May 18, 2012



Update from APHIS Regarding a Detection of Bovine Spongiform Encephalopathy (BSE) in the United States Friday May 18, 2012







Sunday, May 27, 2012



CANADA PLANS TO IMPRISON ANYONE SPEAKING ABOUT MAD COW or ANY OTHER DISEASE OUTBREAK, CENSORSHIP IS A TERRIBLE THING

Tuesday, February 15, 2011

TAIWAN DOH censured for downplaying probable human mad-cow disease death 2011/02/15 18:32:27

DOH censured for downplaying probable human mad-cow disease death 2011/02/15 18:32:27
 
Taipei, Feb. 15 (CNA) The Department of Health (DOH) was censured by the Control Yuan Tuesday for downplaying a probable case of death from Creutzfeldt-Jakob disease (CJD) last year.
 
The top government watchdog said the incident indicates that the DOH lacks a flexible mechanism to guide the release of information on major diseases.
 
The way the DOH handled the incident was questionable because it failed to clear up the truth or disperse people's doubts about the case, the Control Yuan said.
 
A Taiwanese man, who lived in the United Kingdom from 1989 to 1997, died in May 2010 from what appeared to be CJD, a disease linked to eating tissue from cattle infected with bovine spongiform encephalopathy, or mad-cow disease.
 
The patient, who began to show symptoms such as memory loss and hypersomnia, was reported to health authorities as a suspected CJD case in March 2009.
 
While the man's family refused to give permission for an autopsy, a medical team determined that it was an "extremely likely case" of CJD based on his MRI and EEG records.
 
However, the DOH did not publish the details until Dec. 8, 2010, six months after his death and after the case had been reported by local media.
 
According to the Control Yuan, the DOH chose to gloss over the incident because at the time it was first reported to the health authorities in March 2009, Taiwan was negotiating sensitive beef trade issues with the United States.
 
An investigation by the Control Yuan shows that the DOH never notified the National Security Council -- which was leading the Taiwan-U.S. beef trade talks -- about the suspected CJD case.
 
This gave people the impression that the DOH's decision on whether to release disease information was based solely on "
 
 
 
Saturday, December 18, 2010
 
First probable human case of mad cow disease in Taiwan was listed posthumously 2010/12/18 21:14:28
 
 
 
Thursday, November 25, 2010
 
Probable variant Creutzfeldt-Jakob disease in Asia: A case report from Taiwan and review of two prior cases
 
 
 
Tuesday, January 5, 2010
 
JOINT STATEMENT FROM USTR, USDA ON TAIWAN'S ACTIONS TO UNJUSTIFIABLY RESTRICT U.S. BEEF IMPORTS IN VIOLATION OF OUR BILATERAL AGREEMENT Release No. 0002.10 Contact: USTR, Nefeterius McPherson (202) 395-3230 USDA, Caleb Weaver (202) 720-4623
 
 
 
Tuesday, December 29, 2009
 
Taiwan to resume USA beef ban over mad cow disease threat
 
 
 
Saturday, December 18, 2010
 
OIE Global Conference on Wildlife Animal Health and Biodiversity - Preparing for the Future (TSE AND PRIONS) Paris (France), 23-25 February 2011
 
 
 
The most recent assessments (and reassessments) were published in June 2005 (Table I; 18), and included the categorisation of Canada, the USA, and Mexico as GBR III. Although only Canada and the USA have reported cases, the historically open system of trade in North America suggests that it is likely that BSE is present also in Mexico.
 
 
 
Rare BSE mutation raises concerns over risks to public health
 
SIR — Atypical forms (known as H- and L-type) of bovine spongiform encephalopathy (BSE) have recently appeared in several European countries as well as in Japan, Canada and the United States. This raises the unwelcome possibility that variant Creutzfeldt–Jakob disease (vCJD) could increase in the human population. Of the atypical BSE cases tested so far, a mutation in the prion protein gene (PRNP) has been detected in just one, a cow in Alabama with BSE; her healthy calf also carried the mutation (J. A. Richt and S. M. Hall PLoS Pathog. 4, e1000156; 2008). This raises the possibility that the disease could occasionally be genetic in origin. Indeed, the report of the UK BSE Inquiry in 2000 suggested that the UK epidemic had most likely originated from such a mutation and argued against the scrapierelated assumption. Such rare potential pathogenic PRNP mutations could occur in countries at present considered to be free of BSE, such as Australia and New Zealand. So it is important to maintain strict surveillance for BSE in cattle, with rigorous enforcement of the ruminant feed ban (many countries still feed ruminant proteins to pigs). Removal of specified risk material, such as brain and spinal cord, from cattle at slaughter prevents infected material from entering the human food chain. Routine genetic screening of cattle for PRNP mutations, which is now available, could provide additional data on the risk to the public. Because the point mutation identified in the Alabama animals is identical to that responsible for the commonest type of familial (genetic) CJD in humans, it is possible that the resulting infective prion protein might cross the bovine–human species barrier more easily. Patients with vCJD continue to be identified. The fact that this is happening less often should not lead to relaxation of the controls necessary to prevent future outbreaks. Malcolm A. Ferguson-Smith Cambridge University Department of Veterinary Medicine, Madingley Road, Cambridge CB3 0ES, UK e-mail: mhtml:{33B38F65-8D2E-434D-8F9B-8BDCD77D3066}mid://00000026/!x-usc:mailto:maf12@cam.ac.uk J├╝rgen A. Richt College of Veterinary Medicine, Kansas State University, K224B Mosier Hall, Manhattan, Kansas 66506-5601, USA
 
NATURE|Vol 457|26 February 2009
 
 
 
Saturday, January 29, 2011
 
Atypical L-Type Bovine Spongiform Encephalopathy (L-BSE) Transmission to Cynomolgus Macaques, a Non-Human Primate
 
Jpn. J. Infect. Dis., 64 (1), 81-84, 2011
 
 
 
Saturday, June 12, 2010
 
PUBLICATION REQUEST AND FOIA REQUEST Project Number: 3625-32000-086-05 Study of Atypical Bse
 
 
 
Wednesday, July 28, 2010
 
re-Freedom of Information Act Project Number 3625-32000-086-05, Study of Atypical BSE UPDATE July 28, 2010
 
 
 
P.9.21
 
Molecular characterization of BSE in Canada
 
Jianmin Yang1, Sandor Dudas2, Catherine Graham2, Markus Czub3, Tim McAllister1, Stefanie Czub1 1Agriculture and Agri-Food Canada Research Centre, Canada; 2National and OIE BSE Reference Laboratory, Canada; 3University of Calgary, Canada
 
Background: Three BSE types (classical and two atypical) have been identified on the basis of molecular characteristics of the misfolded protein associated with the disease. To date, each of these three types have been detected in Canadian cattle.
 
Objectives: This study was conducted to further characterize the 16 Canadian BSE cases based on the biochemical properties of there associated PrPres. Methods: Immuno-reactivity, molecular weight, glycoform profiles and relative proteinase K sensitivity of the PrPres from each of the 16 confirmed Canadian BSE cases was determined using modified Western blot analysis.
 
Results: Fourteen of the 16 Canadian BSE cases were C type, 1 was H type and 1 was L type. The Canadian H and L-type BSE cases exhibited size shifts and changes in glycosylation similar to other atypical BSE cases. PK digestion under mild and stringent conditions revealed a reduced protease resistance of the atypical cases compared to the C-type cases. N terminal- specific antibodies bound to PrPres from H type but not from C or L type. The C-terminal-specific antibodies resulted in a shift in the glycoform profile and detected a fourth band in the Canadian H-type BSE.
 
Discussion: The C, L and H type BSE cases in Canada exhibit molecular characteristics similar to those described for classical and atypical BSE cases from Europe and Japan. This supports the theory that the importation of BSE contaminated feedstuff is the source of C-type BSE in Canada. *It also suggests a similar cause or source for atypical BSE in these countries.
 
 
 
10,000,000+ LBS. of PROHIBITED BANNED MAD COW FEED I.E. BLOOD LACED MBM IN COMMERCE USA 2007
 
Date: March 21, 2007 at 2:27 pm PST
 
RECALLS AND FIELD CORRECTIONS: VETERINARY MEDICINES -- CLASS II
 
___________________________________
 
PRODUCT
 
Bulk cattle feed made with recalled Darling's 85% Blood Meal, Flash Dried, Recall # V-024-2007
 
CODE
 
Cattle feed delivered between 01/12/2007 and 01/26/2007
 
RECALLING FIRM/MANUFACTURER
 
Pfeiffer, Arno, Inc, Greenbush, WI. by conversation on February 5, 2007.
 
Firm initiated recall is ongoing.
 
REASON
 
Blood meal used to make cattle feed was recalled because it was cross- contaminated with prohibited bovine meat and bone meal that had been manufactured on common equipment and labeling did not bear cautionary BSE statement.
 
VOLUME OF PRODUCT IN COMMERCE
 
42,090 lbs.
 
DISTRIBUTION
 
WI
 
___________________________________
 
PRODUCT
 
Custom dairy premix products: MNM ALL PURPOSE Pellet, HILLSIDE/CDL Prot- Buffer Meal, LEE, M.-CLOSE UP PX Pellet, HIGH DESERT/ GHC LACT Meal, TATARKA, M CUST PROT Meal, SUNRIDGE/CDL PROTEIN Blend, LOURENZO, K PVM DAIRY Meal, DOUBLE B DAIRY/GHC LAC Mineral, WEST PIONT/GHC CLOSEUP Mineral, WEST POINT/GHC LACT Meal, JENKS, J/COMPASS PROTEIN Meal, COPPINI - 8# SPECIAL DAIRY Mix, GULICK, L-LACT Meal (Bulk), TRIPLE J - PROTEIN/LACTATION, ROCK CREEK/GHC MILK Mineral, BETTENCOURT/GHC S.SIDE MK-MN, BETTENCOURT #1/GHC MILK MINR, V&C DAIRY/GHC LACT Meal, VEENSTRA, F/GHC LACT Meal, SMUTNY, A- BYPASS ML W/SMARTA, Recall # V-025-2007
 
CODE
 
The firm does not utilize a code - only shipping documentation with commodity and weights identified.
 
RECALLING FIRM/MANUFACTURER
 
Rangen, Inc, Buhl, ID, by letters on February 13 and 14, 2007. Firm initiated recall is complete.
 
REASON
 
Products manufactured from bulk feed containing blood meal that was cross contaminated with prohibited meat and bone meal and the labeling did not bear cautionary BSE statement.
 
VOLUME OF PRODUCT IN COMMERCE
 
9,997,976 lbs.
 
DISTRIBUTION
 
ID and NV
 
END OF ENFORCEMENT REPORT FOR MARCH 21, 2007
 
 
 
Thursday, November 18, 2010
 
UNITED STATES OF AMERICA VS GALEN J. NIEHUES FAKED MAD COW FEED TEST ON 92 BSE INSPECTION REPORTS FOR APPROXIMATELY 100 CATTLE OPERATIONS
 
 
 
Wednesday, November 17, 2010
 
MAD COW TESTING FAKED IN USA BY Nebraska INSPECTOR Senator Mike Johanns STATE
 
 
 
Tuesday, November 02, 2010
 
BSE - ATYPICAL LESION DISTRIBUTION (RBSE 92-21367) statutory (obex only) diagnostic criteria CVL 1992
 
 
 
Wednesday, July 28, 2010
 
Atypical prion proteins and IBNC in cattle DEFRA project code SE1796 FOIA Final report
 
 
 
IBNC
 
"All of the 15 cattle tested showed that the brains had abnormally accumulated prion protein."
 
Saturday, February 28, 2009
 
NEW RESULTS ON IDIOPATHIC BRAINSTEM NEURONAL CHROMATOLYSIS "All of the 15 cattle tested showed that the brains had abnormally accumulated PrP" 2009
 
SEAC 102/2
 
 
 
Thursday, December 23, 2010
 
Molecular Typing of Protease-Resistant Prion Protein in Transmissible Spongiform Encephalopathies of Small Ruminants, France, 2002–2009 Volume 17, Number 1–January 2011
 
 
 
Monday, November 22, 2010
 
Atypical transmissible spongiform encephalopathies in ruminants: a challenge for disease surveillance and control
 
REVIEW ARTICLES
 
 
 
Saturday, November 6, 2010
 
TAFS1 Position Paper on Position Paper on Relaxation of the Feed Ban in the EU Berne, 2010 TAFS
 
INTERNATIONAL FORUM FOR TRANSMISSIBLE ANIMAL DISEASES AND FOOD SAFETY a non-profit Swiss Foundation
 
 
 
CHRONIC WASTING DISEASE AND IT'S POTENTIAL TO INFECT HUMANS
 
UPDATED DATA ON 2ND CWD STRAIN
 
Wednesday, September 08, 2010
 
CWD PRION CONGRESS SEPTEMBER 8-11 2010
 
 
 
Tuesday, January 25, 2011
 
Generation of a new form of human PrPSc in vitro by inter-species transmission from cervids prions
 
 
 
THEY KNEW THERE WAS A HIGH RISK FACTOR ALMOST 2 DECADES AGO ;
 
CJD9/10022
 
October 1994
 
Mr R.N. Elmhirst Chairman British Deer Farmers Association Holly Lodge Spencers Lane BerksWell Coventry CV7 7BZ
 
Dear Mr Elmhirst,
 
CREUTZFELDT-JAKOB DISEASE (CJD) SURVEILLANCE UNIT REPORT
 
Thank you for your recent letter concerning the publication of the third annual report from the CJD Surveillance Unit. I am sorry that you are dissatisfied with the way in which this report was published.
 
The Surveillance Unit is a completely independant outside body and the Department of Health is committed to publishing their reports as soon as they become available. In the circumstances it is not the practice to circulate the report for comment since the findings of the report would not be amended. In future we can ensure that the British Deer Farmers Association receives a copy of the report in advance of publication.
 
The Chief Medical Officer has undertaken to keep the public fully informed of the results of any research in respect of CJD. This report was entirely the work of the unit and was produced completely independantly of the the Department.
 
The statistical results reqarding the consumption of venison was put into perspective in the body of the report and was not mentioned at all in the press release. Media attention regarding this report was low key but gave a realistic presentation of the statistical findings of the Unit. This approach to publication was successful in that consumption of venison was highlighted only once by the media ie. in the News at one television proqramme.
 
I believe that a further statement about the report, or indeed statistical links between CJD and consumption of venison, would increase, and quite possibly give damaging credence, to the whole issue. From the low key media reports of which I am aware it seems unlikely that venison consumption will suffer adversely, if at all.
 
 
 
CONSUMPTION OF VEAL AND VENISON AND SIGNIFICANT INCREASE CJD
 
Sat Apr 28, 2007 07:10 68.238.100.254
 
greetings,
 
i have been working on something for a while 'the big lie', the following data to be added in, but i thought due to what i think the importance of this is, i thought i would go ahead and put this out now for those that might be interested. ........tss
 
CHANGING SCIENCE TO FIT YOUR INDUSTRY NEEDS COVER-UP IN FULL MODE NOW
 
POLICY - RESTRICTED
 
CREUTZFELDT-JAKOB DISEASE: 3RD ANNUAL REPORT OF THE UK SURVEILLANCE UNIT
 
1. This submission, which has been agreed with colleagues in HEF(M). alerts PS(L) to the contents of the forthcoming annual report of the CJD Surveillance Unit and presents options for publication. It also highlights concern over the presentation of results which could be misrepresented by the media and others as evidence of a lilnk between CJD and the consumption of veal. ...
 
RECOMMENDATION
 
2. PS(L) is invited to agree the recommendation at para 13.
 
PROBLEM
 
7. The main findings in the case-control study were STATISTICALLY SIGNIFICANT ASSOCIATIONS BETWEEN CONSUMPTION OF VEAL OR VENISON AND THE DEVELOPMENT OF CJD (INCREASED RISKS OF 2-13x). There was also evidence of a dose-response relationship between dietary exposure and development of the disease. (Last year's findings showed an apparent association between eating black pudding and risk of CJD which was neither statistically significant nor biologically plausible - interestingly, this has not been (replicated was marked out with something i cannot read), and then this complete sentence was marked through to be replaced ;
 
THIS YEAR'S FINDINGS SHOW A NUMBER OF ASSOCIATIONS BUT THE STRONGEST IS FOR VEAL.
 
IP PS(L) wishes to probe this further we think it best to explain the matter VERBALLY. The problem is how to present the findings in this year's annual report in a way which avoids unnecessary public alarm and limits the scope for media scare stores. (or the facts...TSS)
 
This is of considerable concern given recent development. In particular Ministers will be particularly concerned about the European dimension given the recent troubles with the Germans.
 
9. DH doctors advise - and we understand Dr Wills agrees - that the association the study found between the developments of CJD and veal consumption cannot be regarded as demonstrating a causal relationship or give any reason to change the advice that eating beef and veal is safe. IF PS(L) wishes to probe this further we think it best to explain the matter verbally. The problem is how to present the findings in this year's annual report in a way which avoids unnecessary public alarm and limits the scope for media scare stories.
 
Next steps ...
 
snip... full text ;
 
 
 
PROBLEM
 
7. The main findings in the case-control study were STATISTICALLY SIGNIFICANT ASSOCIATIONS BETWEEN CONSUMPTION OF VEAL OR VENISON AND THE DEVELOPMENT OF CJD (INCREASED RISKS OF 2-13x). There was also evidence of a dose-response relationship between dietary exposure and development of the disease. (Last year's findings showed an apparent association between eating black pudding and risk of CJD which was neither statistically significant nor biologically plausible - interestingly, this has not been (replicated was marked out with something i cannot read), and then this complete sentence was marked through to be replaced ;
 
see watered down report here ;
 
 
 
Friday, February 11, 2011
 
Atypical/Nor98 Scrapie Infectivity in Sheep Peripheral Tissues
 
 
 
Thursday, February 10, 2011
 
TRANSMISSIBLE SPONGIFORM ENCEPHALOPATHY REPORT UPDATE CANADA FEBRUARY 2011 and how to hide mad cow disease in Canada Current as of: 2011-01-31
 
 
 
Friday, February 11, 2011
 
Creutzfeldt-Jakob disease (CJD) biannual update (2010/1) Emerging infections/CJD
 
 
 
 
 
TSS